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J Korean Neurol Assoc. 2005;23(5):682-685.
- Apraxia of Lid Opening after Traumatic Brain Injury: A Delayed Complication
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Sung Gyun Joo
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Department of Neurology, Yeungnam University College of Medicine, Daegu, Korea
- 외상성 뇌손상 후 지연성 후유증으로 발생한 눈뜨기행위상실증 2예
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주성균, 박미영 이 준 조용국 김정현 정지윤
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영남대학교 의과대학 신경과학교실
- Abstract
- Apraxia of lid opening (ALO) has been suggested to be a dysfunction of the supranuclear control of the levator palpebrae superioris caused mainly by basal ganglial lesion. The hypometabolism of the medial frontal lobe may be a pathophysiologic mechanism in ALO. We report two ALO patients who developed these symptoms as a delayed complication after traumatic brain injury (TBI). Their MRI showed encephalomalacia in the Rt. medial frontal cortex, which was not shown in initial brain CT scans. Delayed pathologic changes after TBI may contribute to the development of ALO in these cases. KeyWords:Apraxia of lid opening, Traumatic brain injury, Non dominant frontal lobe
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